Publications for Salomon Amar salomon.amar2@touro.edu

NYMC School of Medicine
Touro College of Dental Medicine at NYMC
Office of the Provost
  • Huck, O., Al-Hashemi, J., Poidevin, L., Poch, O., Davideau, J. L., Tenenbaum, H., & Amar, S. (2017). Identification and characterization of microRNA differentially expressed in macrophages exposed to Porphyromonas gingivalis infection. Infection and Immunity, 85(3), e00771-16. This material can be found here.

  • Cai, B., Panek, J. S., & Amar, S. (2016). Convergent synthesis of novel muramyl dipeptide analogues: Inhibition of porphyromonas gingivalis-induced pro-inflammatory effects by high doses of muramyl dipeptide. Journal of Medicinal Chemistry, 59(14), 6878-6890. doi:10.1021/acs.jmedchem.6b00681

  • Tang, X., & Amar, S. (2016). Kavain involvement in LPS-induced signaling pathways. Journal of Cellular Biochemistry, 117(10), 2272-2280. doi:10.1002/jcb.25525

  • Amar, S., & Engelke, M. (2015). Periodontal innate immune mechanisms relevant to atherosclerosis. Molecular Oral Microbiology, 30(3), 171-185. doi:10.1111/omi.12087

  • Tang, X., & Amar, S. (2015). Kavain inhibition of LPS-induced TNF-α via ERK/LITAF. Toxicology Research, 5(1), 188-196. doi:10.1039/C5TX00164A

  • Tang, X., & Amar, S. (2015). p53 suppresses CCL2-induced subcutaneous tumor xenograft. Tumor Biology, 36(4), 2801-2808. doi:10.1007/s13277-014-2906-9

  • Amar, S., & Leeman, S. (2013). Periodontal innate immune mechanisms relevant to obesity. Molecular Oral Microbiology, 28(5), 331-341. doi:10.1111/omi.12035

  • Bertolo, C., Roa, S., Sagardoy, A., Mena-Varas, M., Robles, E. F., Martinez-Ferrandis, J. I., . . . Amar, S. (2013). LITAF, a BCL6 target gene, regulates autophagy in mature B-cell lymphomas. British Journal of Haemotology, 162(5), 621-630. doi:10.1111/bjh.12440

  • Mazumdar, V., Amar, S., & Segrè, D. (2013). Metabolic proximity in the order of colonization of a microbial community. PLOS One, 8(10) [Article e77617]. doi:10.1371/journal.pone.0077617

  • Richard, G., Trivedi, N., Belta, C., & Amar, S. (2013). Partial restoration of macrophage alteration from diet-induced obesity in response to Porphyromonas gingivalis infection. PLOS One, 8(7) [Article e70320]. doi:10.1371/journal.pone.0070320

  • Tang, X., Yang, Y., Yuan, H., You, J., Burkatovskaya, M., & Amar, S. (2013). Novel transcriptional regulation of VEGF in inflammatory processes. Journal of Cellular and Molecular Medicine, 17(3), 386-397. doi:10.1111/jcmm.12020

  • Treves-Manusevitz, S., Hoz, L., Rachima, H., Montoya, G., Tzur, E., Vardimon, A., . . . Amar, S. (2013). Stem cells of the lamina propria of human oral mucosa and gingiva develop into mineralized tissues in vivo. Journal of Clinical Periodontology, 40(1), 73-81. doi:10.1111/jcpe.12016

  • Yuan, H., Zelka, S., Burkatovskaya, M., Gupte, R., Leeman, S. E., & Amar, S. (2013). Pivotal role of NOD2 in inflammatory processes affecting atherosclerosis and periodontal bone loss. Proceedings of the National Academy of Sciences of the United States of America, 110(52), E5059-E5068. doi:10.1073/pnas.1320862110

  • Brown, B. N., Ratner, B. D., Goodman, S. B., Amar, S., & Badylak, S. F. (2012). Macrophage polarization: An opportunity for improved outcomes in biomaterials and regenerative medicine. Biomaterials, 33(15), 3792-3802. doi:10.1016/j.biomaterials.2012.02.034

  • Liu, B., Faller, L. L., Klitgord, N., Mazumdar, V., Ghodsi, M., Sommer, D. D., . . . Amar, S. (2012). Deep sequencing of the oral microbiome reveals signatures of periodontal disease. PLOS One, 7(6) doi:10.1371/journal.pone.0037919

  • Richard, G., Belta, C., Julius, A. A., & Amar, S. (2012). Controlling the outcome of the toll-like receptor signaling pathways. PLOS One, 7(2) [Article e31341]. doi:10.1371/journal.pone.0031341

  • Tang, X., Asano, M., O'Reilly, A., Farquhar, A., Yang, Y., & Amar, S. (2012). p53 is an important regulator of CCL2 gene expression. Current Molecular Medicine, 12(8), 929-943. doi:10.2174/156652412802480844

  • Bushell, K. N., Leeman, S. E., Gillespie, E., Gower, A. C., Reed, K. L., Stucchi, A. F., . . . Amar, S. (2011). LITAF mediation of increased TNF-α secretion from inflamed colonic lamina propria macrophages. PLOS One, 6(9) [Article e25849]. doi:10.1371/journal.pone.0025849

  • Chang, H., Richard, G., Julius, A. A., Belta, C., & Amar, S. (2011). An application of monotone functions decomposition to the reconstruction of gene regulatory networks. Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society, 2430-2433. doi:10.1109/IEMBS.2011.6090676

  • Merrill, J. C., You, J., Constable, C., Leeman, S. E., & Amar, S. (2011). Whole-body deletion of LPS-induced TNF-α factor (LITAF) markedly improves experimental endotoxic shock and inflammatory arthritis. Proceedings of the National Academy of Sciences of the United States of America, 108(52), 21247-21252. doi:10.1073/pnas.1111492108

  • Richard, G., Chang, H., Cizelj, I., Belta, C., Julius, A. A., & Amar, S. (2011). Integration of large-scale metabolic, signaling, and gene regulatory networks with application to infection responses. Proceedings of the IEEE Conference on Decision and Control, 2227-2232. doi:10.1109/CDC.2011.6160954

  • Tang, X., O'Reilly, A., Asano, M., Merrill, J. C., Yokoyama, K. K., & Amar, S. (2011). P53 peptide prevents LITAF-induced TNF-αlpha-mediated mouse lung lesions and endotoxic shock. Current Molecular Medicine, 11(6), 439-452. This material can be found here.

  • Tang, X., Yang, Y., & Amar, S. (2011). Novel regulation of CCL2 gene expression by murine LITAF and STAT6B. PLOS One, 6(9) [Article e25083]. doi:10.1371/journal.pone.0025083

  • Yuan, H., Gupte, R., Zelkha, S., & Amar, S. (2011). Receptor activator of nuclear factor kappa B ligand antagonists inhibit tissue inflammation and bone loss in experimental periodontitis. Journal of Clinical Periodontology, 38(11), 1029-1036. doi:10.1111/j.1600-051X.2011.01780.x

  • Zhou, J., Yang, Z., Tsuji, T., Gong, J., Xie, J., Chen, C., . . . Amar, S. (2011). LITAF and TNFSF15, two downstream targets of AMPK, exert inhibitory effects on tumor growth. Oncogene, 30(16), 1892-1900. doi:10.1038/onc.2010.575

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