Publications for Pravin B. Sehgal pravin_sehgal@nymc.edu
Sehgal, P. B., Yang, Y. M., & Miller, E. J. (2015). Hypothesis: Neuroendocrine mechanisms (hypothalamus-growth hormone-STAT5 axis) contribute to sex bias in pulmonary hypertension. Molecular Medicine, 21, 688-701. doi:10.2119/molmed.2015.00122
Yang, Y. M., Yuan, H., Edwards, J. G., Skayian, Y., Ochani, K., Miller, E. J., & Sehgal, P. B. (2015). Deletion of STAT5a/b in vascular smooth muscle abrogates the male bias in hypoxic pulmonary hypertension in mice: Implications in the human disease. Molecular Medicine, 20, 625-638. This material can be found here.
Yuan, H., & Sehgal, P. B. (2014). Modulation of transcriptionally productive BMP4-Smad5 signaling along the endocytic pathway by MxA, a dynamin-family GTPase induced by interferon. Molecular Biology of the Cell, 25(25), P1378.
Khan, R., Lee, J. E., Yang, Y. M., Liang, F. X., & Sehgal, P. B. (2013). Live-cell imaging of the association of STAT6-GFP with mitochondria. PLoS One, 8(1) [Article e55426]. doi:10.1371/journal.pone.0055426
Lee, J. E., Yang, Y. M., Yuan, H., & Sehgal, P. B. (2013). Definitive evidence using enucleated cytoplasts for a nongenomic basis for the cystic change in endoplasmic reticulum structure caused by STAT5a/b siRNAs. American Journal of Physiology: Cell Physiology, 304(4), C312-C323. doi:10.1152/ajpcell.00311.2012
Lee, J. E., Yuan, H., Liang, F. X., & Sehgal, P. B. (2013). Nitric oxide scavenging causes remodeling of the endoplasmic reticulum, golgi apparatus and mitochondria in pulmonary arterial endothelial cells. Nitric Oxide: Biology and Chemistry, 33, 64-73. doi:10.1016/j.niox.2013.06.005
Sehgal, P. B. (2013). Non-genomic STAT5-dependent effects at the endoplasmic reticulum and golgi apparatus and STAT6-GFP in mitochondria. Jak-Stat, 2(4) [Article e24860]. doi:10.4161/jkst.24860
Yang, Y. M., Lane, K. B., & Sehgal, P. B. (2013). Subcellular mechanisms in pulmonary arterial hypertension: Combinatorial modalities that inhibit anterograde trafficking and cause bone morphogenetic protein receptor type 2 mislocalization. Pulmonary Circulation, 3(3), 533-550. doi:10.1086/674336
