Faculty Publications
As of August 2017, this database is no longer being updated. For the most current publications from the faculty, students, and staff of Touro University, please check our institutional repository, Touro Scholar, and email any questions or publication submissions to touro.scholar@touro.edu.
Total number of publications: 7,082
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Amar, S., & Engelke, M. (2015). Periodontal innate immune mechanisms relevant to atherosclerosis. Molecular Oral Microbiology, 30(3), 171-185. doi:10.1111/omi.12087
Amar, S., & Leeman, S. (2013). Periodontal innate immune mechanisms relevant to obesity. Molecular Oral Microbiology, 28(5), 331-341. doi:10.1111/omi.12035
Amar, S., Gokce, N., Morgan, S., Loukideli, M., Van Dyke, T. E., & Vita, J. A. (2003). Periodontal disease is associated with brachial artery endothelial dysfunction and systemic inflammation. Arteriosclerosis, Thrombosis, and Vascular Biology, 23(7), 1245-1249. doi:10.1161/01.ATV.0000078603.90302.4A
Amar, S., Oyaisu, K., Li, L., & Van Dyke, T. (2001). Moesin: A potential LPS receptor on human monocytes. Journal of Endotoxin Research, 7(4), 281-286. doi:10.1179/096805101101532855
Amar, S., Petrungaro, P., Amar, A., & Van Dyke, T. E. (1995). Immunolocalization of bone matrix macromolecules in human tissues regenerated from periodontal defects treated with expanded polytetrafluoroethylene membranes. Archives of Oral Biology, 40(7), 653-661. doi:10.1016/0003-9969(94)00201-L
Amar, S., Van Dyke, T. E., Eugster, H. P., Schultze, N., Koebel, P., & Bluethmann, H. (1995). Tumor necrosis factor (TNF)-lnduced cutaneous necrosis is mediated by TNF receptor. Journal of Inflammation, 47(4), 180-189. This material can be found here.
Amar, S., Wu, S. C., & Madan, M. (2009). Is porphyromonas gingivalis cell invasion required for atherogenesis? Pharmacotherapeutic implications. Journal of Immunology, 182(3), 1584-1592. This material can be found here.
Amar, S., Zhou, Q., Shaik-Dasthagirisaheb, Y., & Leeman, S. (2007). Diet-induced obesity in mice causes changes in immune responses and bone loss manifested by bacterial challenge. Proceedings of the National Academy of Sciences of the United States of America, 104(51), 20466-20471. doi:10.1073/pnas.0710335105
Assuma, R., Oates, T., Cochran, D., Amar, S., & Graves, D. T. (1998). IL-1 and TNF antagonists inhibit the inflammatory response and bone loss in experimental periodontitis. Journal of Immunology, 160(1), 403-409. This material can be found here.
Bolcato-Bellemin, A. L., Mattei, M. G., Fenton, M., & Amar, S. (2004). Molecular cloning and characterization of mouse LITAF cDNA: Role in the regulation of tumor necrosis factor-α (TNF-α) gene expression. Journal of Endotoxin Research, 10(1), 15-23. doi:10.1179/096805104225003780
Bushell, K. N., Leeman, S. E., Gillespie, E., Gower, A. C., Reed, K. L., Stucchi, A. F., . . . Amar, S. (2011). LITAF mediation of increased TNF-α secretion from inflamed colonic lamina propria macrophages. PLOS One, 6(9) [Article e25849]. doi:10.1371/journal.pone.0025849
Cai, B., Panek, J. S., & Amar, S. (2016). Convergent synthesis of novel muramyl dipeptide analogues: Inhibition of porphyromonas gingivalis-induced pro-inflammatory effects by high doses of muramyl dipeptide. Journal of Medicinal Chemistry, 59(14), 6878-6890. doi:10.1021/acs.jmedchem.6b00681
Chi, H., Messas, E., Levine, R. A., Graves, D. T., & Amar, S. (2004). Interleukin-1 receptor signaling mediates atherosclerosis associated with bacterial exposure and/or a high-fat diet in a murine apolipoprotein E heterozygote model: Pharmacotherapeutic implications. Circulation, 110(12), 1678-1685. doi:10.1161/01.CIR.0000142085.39015.31
Chiang, C. Y., Kyritsis, G., Graves, D. T., & Amar, S. (1999). Interleukin-1 and tumor necrosis factor activities partially account for calvarial bone resorption induced by local injection of lipopolysaccharide. Infection and Immunity, 67(8), 4231-4236. This material can be found here.
Delima, A. J., Karatzas, S., Amar, S., & Graves, D. T. (2002). Inflammation and tissue loss caused by periodontal pathogens is reduced by interleukin-1 antagonists. Journal of Infectious Diseases, 186(4), 511-516. doi:10.1086/341778
Delima, A. J., Oates, T., Assuma, R., Schwartz, Z., Cochran, D., Amar, S., & Graves, D. T. (2001). Soluble antagonists to interleukin-1 (IL-1) and tumor necrosis factor (TNF) inhibits loss of tissue attachment in experimental periodontitis. Journal of Clinical Periodontology, 28(3), 233-240. doi:10.1034/j.1600-051x.2001.028003233.x
Genco, C. A., Van Dyke, T., & Amar, S. (1998). Animal models for porphyromonas gingivalis-mediated periodontal disease. Trends in Microbiology, 6(11), 444-449. doi:10.1016/S0966-842X(98)01363-8
Graves, D. T., Delima, A. J., Assuma, R., Amar, S., Oates, T., & Cochran, D. (1998). Interleukin-1 and tumor necrosis factor antagonists inhibit the progression of inflammatory cell infiltration toward alveolar bone in experimental periodontitis. Journal of Periodontology, 69(12), 1419-1425.
Graves, D. T., Naguib, G., Lu, H., Desta, T., & Amar, S. (2005). Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis. Journal of Endotoxin Research, 11(1), 13-18. doi:10.1179/096805105225006722
Graves, D. T., Nooh, N., Gillen, T., Davey, M., Patel, S., Cottrell, D., & Amar, S. (2001). IL-1 plays a critical role in oral, but not dermal, wound healing. Journal of Immunology, 167(9), 5316-5320. This material can be found here.
Han, X., & Amar, S. (2003). Role of insulin-like growth factor-1 signaling in dental fibroblast apoptosis. Journal of Periodontology, 74(8), 1176-1182. doi:10.1902/jop.2003.74.8.1176
Hoge, M., & Amar, S. (2006). Role of interleukin-1 in bacterial atherogenesis. Drugs of Today, 42(10), 683-688. doi:10.1358/dot.2006.42.10.1003543
Iontcheva, I., Amar, S., Zawawi, K. H., Kantarci, A., & Van Dyke, T. E. (2004). Role for moesin in lipopolysaccharide-stimulated signal transduction. Infection and Immunity, 72(4), 2312-2320. doi:10.1128/IAI.72.4.2312-2320.2004
Karatzas, S., Zavras, A., Greenspan, D., & Amar, S. (1999). Histologic observations of periodontal wound healing after treatment with PerioGlas in nonhuman primates. International Journal of Periodontics and Restorative Dentistry, 19(5), 489-499. This material can be found here.
Landi, L., Amar, S., Polins, A. S., & Van Dyke, T. E. (1997). Host mechanisms in the pathogenesis of periodontal disease. Current Opinion in Periodontology, 4, 3-10. This material can be found here.
