Faculty Publications
As of August 2017, this database is no longer being updated. For the most current publications from the faculty, students, and staff of Touro University, please check our institutional repository, Touro Scholar, and email any questions or publication submissions to touro.scholar@touro.edu.
Total number of publications: 7,082
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Yum, M. S., Lee, M., Ko, T. S., & Velisek, L. (2014). A potential effect of ganaxolone in an animal model of infantile spasms. Epilepsy Research, 108(9), 1492-1500. doi:10.1016/j.eplepsyres.2014.08.015
Yum, M. S., Lee, M., Woo, D. C., Kim, D. W., Ko, T. S., & Velisek, L. (2015). Beta-hydroxybutyrate attenuates NMDA-induced spasms in rats with evidence of neuronal stabilization on MR spectroscopy. Epilepsy Research, 117, 125-132. doi:10.1016/j.eplepsyres.2015.08.005
Zhang, X. L., Guariglia, S. R., McGlothan, J. L., Stansfield, K. H., Stanton, P. K., & Guilarte, T. R. (2015). Presynaptic mechanisms of lead neurotoxicity: Effects on vesicular release, vesicle clustering and mitochondria number. PLoS One, 10(5), [Article e0127461]. doi:10.1371/journal.pone.0127461
Zhang, X. L., Poschel, B., Faul, C., Upreti, C., Stanton, P. K., & Mundel, P. (2013). Essential role for synaptopodin in dendritic spine plasticity of the developing hippocampus. The Journal of Neuroscience, 33(30), 12510-12518. doi:10.1523/JNEUROSCI.2983-12.2013
Zhang, X. L., Shuttleworth, C. W., Moskal, J. R., & Stanton, P. K. (2015). Suppression of spreading depolarization and stabilization of dendritic spines by GLYX-13, an NMDA receptor glycine-site functional partial agonist. Experimental Neurology, 276, 312-321. doi:10.1016/j.expneurol.2015.07.025
Zhang, Y. T., Alber, M. S., & Newman, S. A. (2013). Mathematical modeling of vertebrate limb development. Mathematical Biosciences, 243(1), 1-17. doi:10.1016/j.mbs.2012.11.003
Zia, M. T., Vinukonda, G., Vose, L. R., Iacobas, S., La Gamma, E. F., Iacobas, D. A., . . . Ballabh, P. (2015). Postnatal glucocorticoid-induced hypomyelination, gliosis, and neurologic deficits are dose-dependent, preparation-specific, and reversible. Experimental Neurology, 263, 200-213. doi:10.1016/j.expneurol.2014.09.013
